Luciano D'Adamio

academic, NJMS-Pharmacology

Emailld550@rutgers.edu

Source: Scopus
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1 Active

Studies of dementia pathogenesis in genetically faithful rat models of Familal Alzheimer disease
D'Adamio, L. & D'ADAMIO, L.
National Institute on Aging
4/1/19 → 1/31/22
Project: Research project

6723 Citations
43 h-Index
104 Article
3 Comment/debate
3 Review article

Knock-in rats with homozygous PSEN1^L435F Alzheimer mutation are viable and show selective γ-secretase activity loss causing low Aβ40/42 and high Aβ43
Tambini, M. D. & D'Adamio, L., May 22 2020, In: Journal of Biological Chemistry. 295, 21, p. 7442-7451 10 p.
Research output: Contribution to journal › Article › peer-review

Open Access
3 Scopus citations
Microglia TREM2^R47H alzheimer-linked variant enhances excitatory transmission and reduces LTP via increased TNF-α levels
Ren, S., Yao, W., Tambini, M. D., Yin, T., Norris, K. A. & D’adamio, L., Jun 2020, In: eLife. 9, p. 1-18 18 p., e57513.
Research output: Contribution to journal › Article › peer-review

Open Access
3 Scopus citations
Opposite changes in APP processing and human aβ levels in rats carrying either a protective or a pathogenic APP mutation
Tambini, M. D., Norris, K. A. & D’Adamio, L., Feb 2020, In: eLife. 9, e52612.
Research output: Contribution to journal › Article › peer-review

Open Access
6 Scopus citations
Tau is not necessary for amyloid-ß-induced synaptic and memory impairments
Puzzo, D., Argyrousi, E. K., Staniszewski, A., Zhang, H., Calcagno, E., Zuccarello, E., Acquarone, E., Fa, M., Puma, D. D. L., Grassi, C., D'Adamio, L., Kanaan, N. M., Fraser, P. E. & Arancio, O., Sep 1 2020, In: Journal of Clinical Investigation. 130, 9, p. 4831-4844 14 p.
Research output: Contribution to journal › Article › peer-review
3 Scopus citations
Trem2 Splicing and Expression are Preserved in a Human Aβ-producing, Rat Knock-in Model of Trem2-R47H Alzheimer’s Risk Variant
Tambini, M. D. & D’Adamio, L., Dec 1 2020, In: Scientific reports. 10, 1, 4122.
Research output: Contribution to journal › Article › peer-review

Open Access
4 Scopus citations

Studies of dementia pathogenesis in genetically faithful rat models of Familal Alzheimer disease

Knock-in rats with homozygous PSEN1L435F Alzheimer mutation are viable and show selective γ-secretase activity loss causing low Aβ40/42 and high Aβ43

Microglia TREM2R47H alzheimer-linked variant enhances excitatory transmission and reduces LTP via increased TNF-α levels

Opposite changes in APP processing and human aβ levels in rats carrying either a protective or a pathogenic APP mutation

Tau is not necessary for amyloid-ß-induced synaptic and memory impairments

Trem2 Splicing and Expression are Preserved in a Human Aβ-producing, Rat Knock-in Model of Trem2-R47H Alzheimer’s Risk Variant

Knock-in rats with homozygous PSEN1^L435F Alzheimer mutation are viable and show selective γ-secretase activity loss causing low Aβ40/42 and high Aβ43

Microglia TREM2^R47H alzheimer-linked variant enhances excitatory transmission and reduces LTP via increased TNF-α levels