Luciano D'Adamio

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Trem2-mediated microglia-neuronal axis in Alzheimer disease pathogenesis
D'Adamio, L.
8/1/21 → 4/30/22
Project: Research project

111 Article
3 Comment/debate
3 Review article

Initial assessment of the spatial learning, reversal, and sequencing task capabilities of knock-in rats with humanizing mutations in the Aβ-coding region of App
Pham, H., Yin, T. & D’Adamio, L., May 2022, In: PloS one. 17, 5 May, e0263546.
Research output: Contribution to journal › Article › peer-review

Open Access
2 Scopus citations
Late-long-term potentiation magnitude, but not Aβ levels and amyloid pathology, is associated with behavioral performance in a rat knock-in model of Alzheimer disease
Yesiltepe, M., Yin, T., Tambini, M. D., Breuillaud, L., Zehntner, S. P. & D’Adamio, L., Nov 11 2022, In: Frontiers in Aging Neuroscience. 14, 1040576.
Research output: Contribution to journal › Article › peer-review

Open Access
A familial Danish dementia rat shows impaired presynaptic and postsynaptic glutamatergic transmission
Yin, T., Yao, W., Norris, K. A. & D'Adamio, L., Sep 1 2021, In: Journal of Biological Chemistry. 297, 3, 101089.
Research output: Contribution to journal › Article › peer-review

Open Access
2 Scopus citations
Danish and British dementia ITM2b/BRI2 mutations reduce BRI2 protein stability and impair glutamatergic synaptic transmission
Yin, T., Yao, W., Lemenze, A. D. & D'Adamio, L., Jan 1 2021, In: Journal of Biological Chemistry. 296, 100054.
Research output: Contribution to journal › Article › peer-review

Open Access
7 Scopus citations
TNF-α-mediated reduction in inhibitory neurotransmission precedes sporadic Alzheimer's disease pathology in young Trem2R47H rats
Ren, S., Breuillaud, L., Yao, W., Yin, T., Norris, K. A., Zehntner, S. P. & D'Adamio, L., Jan 1 2021, In: Journal of Biological Chemistry. 296, 100089.
Research output: Contribution to journal › Article › peer-review

Open Access
13 Scopus citations

Luciano D'Adamio

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Trem2-mediated microglia-neuronal axis in Alzheimer disease pathogenesis

Initial assessment of the spatial learning, reversal, and sequencing task capabilities of knock-in rats with humanizing mutations in the Aβ-coding region of App

Late-long-term potentiation magnitude, but not Aβ levels and amyloid pathology, is associated with behavioral performance in a rat knock-in model of Alzheimer disease

A familial Danish dementia rat shows impaired presynaptic and postsynaptic glutamatergic transmission

Danish and British dementia ITM2b/BRI2 mutations reduce BRI2 protein stability and impair glutamatergic synaptic transmission

TNF-α-mediated reduction in inhibitory neurotransmission precedes sporadic Alzheimer's disease pathology in young Trem2R47H rats

Luciano D'Adamio

Fingerprint

Network

Projects

Trem2-mediated microglia-neuronal axis in Alzheimer disease pathogenesis

Research output

Initial assessment of the spatial learning, reversal, and sequencing task capabilities of knock-in rats with humanizing mutations in the Aβ-coding region of App

Late-long-term potentiation magnitude, but not Aβ levels and amyloid pathology, is associated with behavioral performance in a rat knock-in model of Alzheimer disease

A familial Danish dementia rat shows impaired presynaptic and postsynaptic glutamatergic transmission

Danish and British dementia ITM2b/BRI2 mutations reduce BRI2 protein stability and impair glutamatergic synaptic transmission

TNF-α-mediated reduction in inhibitory neurotransmission precedes sporadic Alzheimer's disease pathology in young Trem2R47H rats