Dangerous liaisons: Fanconi anemia and toxic nonhomologous end joining in DNA crosslink repair

Samuel F. Bunting; André Nussenzweig

doi:10.1016/j.molcel.2010.07.016

Dangerous liaisons: Fanconi anemia and toxic nonhomologous end joining in DNA crosslink repair

Samuel F. Bunting, André Nussenzweig

Research output: Contribution to journal › Short survey › peer-review

23 Scopus citations

Abstract

The proper choice of repair pathway is critical to tolerating various types of DNA damage. In a recent issue of Molecular Cell, Adamo et al. (2010), along with a second report (Pace et al., 2010), describe how the Fanconi anemia (FA) pathway is involved in preventing aberrant DNA repair. These studies suggest a potentially significant new opportunity for the treatment of FA.

Original language	English (US)
Pages (from-to)	164-166
Number of pages	3
Journal	Molecular cell
Volume	39
Issue number	2
DOIs	https://doi.org/10.1016/j.molcel.2010.07.016
State	Published - Jul 2010
Externally published	Yes

All Science Journal Classification (ASJC) codes

Molecular Biology
Cell Biology

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10.1016/j.molcel.2010.07.016

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title = "Dangerous liaisons: Fanconi anemia and toxic nonhomologous end joining in DNA crosslink repair",

abstract = "The proper choice of repair pathway is critical to tolerating various types of DNA damage. In a recent issue of Molecular Cell, Adamo et al. (2010), along with a second report (Pace et al., 2010), describe how the Fanconi anemia (FA) pathway is involved in preventing aberrant DNA repair. These studies suggest a potentially significant new opportunity for the treatment of FA.",

author = "Bunting, {Samuel F.} and Andr{\'e} Nussenzweig",

year = "2010",

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AU - Nussenzweig, André

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AB - The proper choice of repair pathway is critical to tolerating various types of DNA damage. In a recent issue of Molecular Cell, Adamo et al. (2010), along with a second report (Pace et al., 2010), describe how the Fanconi anemia (FA) pathway is involved in preventing aberrant DNA repair. These studies suggest a potentially significant new opportunity for the treatment of FA.

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Dangerous liaisons: Fanconi anemia and toxic nonhomologous end joining in DNA crosslink repair

Abstract

All Science Journal Classification (ASJC) codes

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