Dangerous liaisons: Fanconi anemia and toxic nonhomologous end joining in DNA crosslink repair

Samuel F. Bunting, André Nussenzweig

Research output: Contribution to journalShort surveypeer-review

23 Scopus citations

Abstract

The proper choice of repair pathway is critical to tolerating various types of DNA damage. In a recent issue of Molecular Cell, Adamo et al. (2010), along with a second report (Pace et al., 2010), describe how the Fanconi anemia (FA) pathway is involved in preventing aberrant DNA repair. These studies suggest a potentially significant new opportunity for the treatment of FA.

Original languageEnglish (US)
Pages (from-to)164-166
Number of pages3
JournalMolecular cell
Volume39
Issue number2
DOIs
StatePublished - Jul 2010
Externally publishedYes

All Science Journal Classification (ASJC) codes

  • Molecular Biology
  • Cell Biology

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