Epidemiological, histopathological and biochemical evidence indicates that AMD is associated with oxidative damage, lipofuscin accumulation, chronic inflammation, and mutations in the complement system. Molecular targets have been identified that may serve as the basis for developing new, better treatments for AMD including prophylactic therapy and treatments for the late stage complications of geographic atrophy and choroidal neovascularization.
|Original language||English (US)|
|Title of host publication||Medical Retina|
|Publisher||S. Karger AG|
|Number of pages||9|
|State||Published - May 24 2012|
All Science Journal Classification (ASJC) codes
- Biochemistry, Genetics and Molecular Biology(all)