Splicing factor hnRNPH drives an oncogenic splicing switch in gliomas

Keyphrases

• Glioma 100%
• Splicing Factor 100%
• Death Domain 100%
• Splicing Switch 100%
• Apoptosis 66%
• Mitogen-activated Protein Kinase 66%
• Invasiveness 66%
• Glucagonoma 66%
• Stem Cells 33%
• Ablation 33%
• Tumor Progression 33%
• Tumor 33%
• Knockdown 33%
• Cell Death 33%
• Aggressive Tumor 33%
• Adaptor Protein 33%
• Anti-apoptotic 33%
• Redirection 33%
• Splicing Event 33%
• Constitutively Active 33%
• Multiple Aspects 33%
• Receptor Subtypes 33%
• Exon 33%
• Tyrosine Kinase Receptor 33%
• Exon 11 33%
• Death Signals 33%
• Glioblastoma multiforme 33%
• Isoform-specific 33%
• Cell Pattern 33%
• Ligand-independent 33%
• Tumor Maintenance 33%
• Tumor Behavior 33%
• Tumor Establishment 33%
• Multiple Keys 33%
• Malignant Glioma 33%
• Aberrant Splicing 33%

Biochemistry, Genetics and Molecular Biology

• Splicing Factor 100%
• Death Domain 100%
• Isoform 66%
• Mitogen-Activated Protein Kinase 66%
• Exon 66%
• Programmed Cell Death 66%
• Downregulation 33%
• Stem Cell 33%
• Cell Death 33%
• Tumor Necrosis Factor 33%
• TRAIL 33%
• Tyrosine Kinase Receptor 33%