XAging well with a little wine and a good clock

William J. Belden; Jay C. Dunlap

doi:10.1016/j.cell.2013.05.055

XAging well with a little wine and a good clock

William J. Belden, Jay C. Dunlap

Research output: Contribution to journal › Short survey › peer-review

6 Scopus citations

Abstract

Age-related decline in mammalian circadian rhythm has been recognized for decades, but the underlying molecular mechanisms have remained elusive. In this issue of Cell, Chang and Guarente use brain-specific SIRT1 knockout mice and transgenic mice overexpressing SIRT1 to develop an enticing model for how SIRT1 helps maintain the robustness of the aging circadian clock.

Original language	English (US)
Pages (from-to)	1421
Number of pages	1
Journal	Cell
Volume	153
Issue number	7
DOIs	https://doi.org/10.1016/j.cell.2013.05.055
State	Published - Jun 20 2013

All Science Journal Classification (ASJC) codes

Biochemistry, Genetics and Molecular Biology(all)

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10.1016/j.cell.2013.05.055

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title = "XAging well with a little wine and a good clock",

abstract = "Age-related decline in mammalian circadian rhythm has been recognized for decades, but the underlying molecular mechanisms have remained elusive. In this issue of Cell, Chang and Guarente use brain-specific SIRT1 knockout mice and transgenic mice overexpressing SIRT1 to develop an enticing model for how SIRT1 helps maintain the robustness of the aging circadian clock.",

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AU - Belden, William J.

AU - Dunlap, Jay C.

PY - 2013/6/20

Y1 - 2013/6/20

N2 - Age-related decline in mammalian circadian rhythm has been recognized for decades, but the underlying molecular mechanisms have remained elusive. In this issue of Cell, Chang and Guarente use brain-specific SIRT1 knockout mice and transgenic mice overexpressing SIRT1 to develop an enticing model for how SIRT1 helps maintain the robustness of the aging circadian clock.

AB - Age-related decline in mammalian circadian rhythm has been recognized for decades, but the underlying molecular mechanisms have remained elusive. In this issue of Cell, Chang and Guarente use brain-specific SIRT1 knockout mice and transgenic mice overexpressing SIRT1 to develop an enticing model for how SIRT1 helps maintain the robustness of the aging circadian clock.

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DO - 10.1016/j.cell.2013.05.055

M3 - Short survey

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VL - 153

SP - 1421

JO - Cell

JF - Cell

IS - 7

ER -

XAging well with a little wine and a good clock

Abstract

All Science Journal Classification (ASJC) codes

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